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dc.contributor.authorAnzinger, JJ
dc.contributor.authorButterfield, TR
dc.contributor.authorAngelovich, TA
dc.contributor.authorCrowe, SM
dc.contributor.authorPalmer, CS
dc.date.accessioned2021-02-12T00:27:31Z
dc.date.available2021-02-12T00:27:31Z
dc.date.issued2014-01-01
dc.identifier.citationAnzinger, J. J., Butterfield, T. R., Angelovich, T. A., Crowe, S. M. & Palmer, C. S. (2014). Monocytes as Regulators of Inflammation and HIV-Related Comorbidities during cART. JOURNAL OF IMMUNOLOGY RESEARCH, 2014, https://doi.org/10.1155/2014/569819.
dc.identifier.issn2314-8861
dc.identifier.urihttp://hdl.handle.net/11343/260611
dc.description.abstractCombined antiretroviral therapy (cART) extends the lifespan and the quality of life for HIV-infected persons but does not completely eliminate chronic immune activation and inflammation. The low level of chronic immune activation persisting during cART-treated HIV infection is associated with the development of diseases which usually occur in the elderly. Although T-cell activation has been extensively examined in the context of cART-treated HIV infection, monocyte activation is only beginning to be recognized as an important source of inflammation in this context. Here we examine markers and sources of monocyte activation during cART-treated HIV infection and discuss the role of monocytes during cardiovascular disease, HIV-associated neurocognitive disorder, and innate immune aging.
dc.languageEnglish
dc.publisherHINDAWI LTD
dc.rights.urihttps://creativecommons.org/licenses/by/4.0
dc.titleMonocytes as Regulators of Inflammation and HIV-Related Comorbidities during cART
dc.typeJournal Article
dc.identifier.doi10.1155/2014/569819
melbourne.affiliation.departmentMicrobiology and Immunology
melbourne.affiliation.facultyMedicine, Dentistry & Health Sciences
melbourne.source.titleJournal of Immunology Research
melbourne.source.volume2014
dc.rights.licenseCC BY
melbourne.elementsid1110103
melbourne.contributor.authorPalmer, Clovis
dc.identifier.eissn2314-7156
melbourne.accessrightsOpen Access


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