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dc.contributor.authorPalma-Rigo, Kesiaen_US
dc.contributor.authorBASSI, JASPREETen_US
dc.contributor.authorNguyen-Huu, Thu-Phucen_US
dc.contributor.authorJackson, Kristy L.en_US
dc.contributor.authorDavern, Pamela J.en_US
dc.contributor.authorCHEN, DAIANen_US
dc.contributor.authorElghozi, Jean-Lucen_US
dc.contributor.authorThomas, Walter Gen_US
dc.contributor.authorALLEN, ANDREWen_US
dc.contributor.authorHead, Geoffrey A.en_US
dc.date.accessioned2014-05-22T07:12:27Z
dc.date.available2014-05-22T07:12:27Z
dc.date.issued2012en_US
dc.identifier0008-6363en_US
dc.identifier.citationPalma-Rigo, K., Bassi, J., Nguyen-Huu, T., Jackson, K. L., Davern, P. J., CHEN, D., et al. (2012). Angiotensin 1A receptors transfected into caudal ventrolateral medulla inhibit baroreflex gain and stress responses. Cardiovascular Research, 96(2).en_US
dc.identifier.urihttp://hdl.handle.net/11343/32833
dc.descriptionC1 - Journal Articles Refereed
dc.formatapplication/pdfen_US
dc.subjectCardiology (incl. Cardiovascular Diseases); Autonomic Nervous System; Expanding Knowledge in the Medical and Health Sciencesen_US
dc.titleAngiotensin 1A receptors transfected into caudal ventrolateral medulla inhibit baroreflex gain and stress responsesen_US
dc.typeJournal Articleen_US
melbourne.peerreviewPeer Revieweden_US
melbourne.affiliationThe University of Melbourneen_US
melbourne.affiliation.departmentPhysiologyen_US
melbourne.source.titleCardiovascular Researchen_US
melbourne.source.volume96en_US
melbourne.source.issue2en_US
melbourne.publicationid185379
melbourne.elementsidNA
melbourne.contributor.authorBassi, Jaspreet
melbourne.contributor.authorCHEN, DAIAN
melbourne.contributor.authorAllen, Andrew
melbourne.fieldofresearch320101 Cardiology (incl. cardiovascular diseases)
melbourne.fieldofresearch320901 Autonomic nervous system
melbourne.seocode280103 Expanding knowledge in the biomedical and clinical science
melbourne.accessrightsThis item is currently not available from this repository


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